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Warts Skin

Warts – infectious benign skin growths rough, having a form of nodules or papillae. The surface elevation is rough, rough, cracked, which viewed the tiny red dots – blood vessels. Ali Partovi: the source for more info. Warts are most often painted in a yellowish-gray color, they have no skin line. Warts, as well as papillomas, warts, molluscum contagiosum, a viral disease. There are several types warts: common, flat, plantar, genital, senile. The most frequent common warts – small knots of diameter 3 – 10 mm. Area of their occurrence – hands and fingers, soles, at least – face and neck.

Flat, or Youth, warts have a diameter of 0.5 – 3 mm, they are teams and there are mostly young people. Plantar warts occur on the feet or areas of maximum pressure of shoes, they often make it difficult to walk, bodies. Senile warts are formed in people older than 40 years, most often situated groups, their number may grow. Preventing warts. If you do not have these tumors, especially prevention for you relevant. If you have one or more of the warts, you also need prevention, that their number has not increased.

Thus, the wart – a viral, rather contagious skin disease. Pathogenic virus is easily penetrates into the body through a break in the skin – cuts, scratches and even hangnail. Cause the spread of infection can damage the warts. Neoplasm does not appear immediately, growing only after 2-5 months after infection. To self-infection has not occurred should observe the following rules: 1. if you have warts in areas that you normally shave (legs, underarms, bikini area), carry out the procedure carefully, without touching warts 2. if you have warts in okolonogtevogo area, and you have a habit of biting his nails, immediately discard this disastrous occupation. Otherwise, you risk increasing the number of warts on the hands 3. Warts better develop in a moist environment.

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NUC Patients

The data obtained in long-term study allowed the following conclusions: – PSC can develop at any stage of the course of IBD, but the majority of patients it occurs within 5 years of onset (80.6% in the NUC, 76.5% in CD) – Lost liver during the first year of onset is more common in CD than in NUC (29.4% in CD, 19.4% at NUC) – The total defeat of the colon in patients with PSC is observed at NUC in 86% of cases, in BC 100% – The course of PSC is independent on the activity of inflammation in the gut. In 10-25% of cases the disease is asymptomatic. Despite the satisfactory state of patients for many years, the disease can progress with the development of cirrhosis liver, and the reason for the survey supports portal hypertension. High risk of PSC in IBD requires mandatory blood testing of biochemical parameters with the definition cholestasis in all patients with NUC and BC, and in its identification – the use of complex diagnostic tests to exclude PSC. Cholangiocellular carcinoma. Adenocarcinoma of the bile duct occurs in 1.5% of patients with NUC and 10% of patients PSC. In patients with NUC risk of cholangiocellular carcinoma is 20 times and seen 10 years earlier than people without IBD.

Diagnosis is often difficult, because the data ERCP is not always distinct from those with PSC. Fatty liver. The frequency of hepatic steatosis in patients with CD and NUC can reach 50%. Clinically, the disease is asymptomatic. It is believed that it should not be regarded as extraintestinal manifestation of IBD, and as a complication of corticosteroid therapy, the result of parenteral nutrition, malabsorption syndrome, sepsis. Autoimmune hepatitis. Autoimmune hepatitis is found in 1-5% of patients with IBD. When he NUC is more common than in CD. In patients with a combination of autoimmune disease and IBD autoimmune nature of the disease is confirmed by the detection of antinuclear antibodies, antibodies to smooth muscle, as well as the identification of other autoimmune diseases.